At the bedside question bank

At the bedside

There are some things you will need to know when tending to a patient at their bedside. Observing a patient’s features and then diagnosing a potential condition is a key skill of the role. This question bank is designed to test your knowledge of all things observation and bedside related.

Just navigate to the question you want or scroll to take the entire test. Once you have guessed, click reveal to show the answer,

Cannula sizes

Order the cannula colour with the correct size (G or gauge) from smallest to largest. Remember the lower the gauge, the wider bore the cannula.

So… it goes from smallest to largest: yellow, blue, pink, green, white, grey, orange. I’d be interested to know how this came about. There’s no clear logic to it!

Signs of chronic liver disease

The following are a sign of / associated with chronic liver disease. For each one, choose true or false?

  • Leuconychia
  • Keiser Fleisher rings
  • Encephalopathy
  • Diabetes mellitus and CLD
  • Gottrons papules
  • Dupytrens

Signs of CLD include:

Leuconychia, kolionychia, asterixis, palmar erythema, pruritis, Dupytrens, bruising, thin skin, ascites, elevated JVP, spider naevi, ascites, jaundice, encephalopathy, hepatomegaly, splenomegaly, peripheral oedema etc etc

Keiser Fleisher rings: Wilsons disease
Diabetes mellitus and CLD: think Haemochromatosis
SOB and CLD: Think Alpha 1 antitrypsin
Female and CLD and no alcohol: PBC and PSC

Gottrons papules are the papules on the backs of the fingers seen in dermatomyositis.

Auer rods are seen on a blood film and are characteristic of AML.

A McMurray test is the testing to look for damaged menisci in the knee examination.

Endocrine observation and diagnosis

For each following observation, what would be your potential diagnosis?

  • Random blood sugar 21, urine: ketones, ABG: pH 7.32
  • Random blood sugar 21, urine: no ketones, ABG: pH 7.42
  • Clinic Blood glucose 4.5 mmol/l, HBA1c 15% [<6.5%]
  • MCV 102 (80-99), CK 240 (<150)
  • Clinic Blood glucose 5.2 mmol/l, HBA1c 6.2%
  • Lid Lag, Lid retraction, goitre
  • Hypothyroidism, no goitre
  • Hypertension, striae, moon face
  • Extreme episodic palpitations with hypertension
  • Excessive thirst, normal blood sugar and elecrolytes
  • Large tongue, prominent supra orbital ridge, changing facial appearance
  • Paroxsysmal flushing of the face and weight loss
  • Gynecomastia
  • Pigmented palmar creases, hypothermia
  • Goitre with hypothyroidism

Observation

Diagnosis

Random blood sugar 21, urine: ketones, ABG: pH 7.32

Probable type 1 diabetes: acidosis and ketosis suggest insulin lack and diabetic ketoacidosis. Seen almost always in type 1 and not type 2 diabetes

Random blood sugar 21, urine: no ketones, ABG: pH 7.42

Probable Type 2 diabetes: Lack of acidosis and normal pH suggest there is a degree of insulin action

Clinic Blood glucose 4.5 mmol/l, HBA1c 15% [<6.5%]

Diabetes mellitus, poor long term glycaemic control: HBA1c reflects glycosylated haemoglobin and thus DM control over the last few months

MCV 102 (80-99), CK 240 (<150)

Hypothyroidism: watch for the high CK due to abnormalities in the muscle cell membrane. Macrocytosis is commonly caused by Hypo/hyperthroidism, B12 deficiency and alcoholism

Clinic Blood glucose 5.2 mmol/l, HBA1c 6.2%

Diabetes mellitus, good long term glycaemic control: HBA1c reflects glycosylated haemaglobin and thus shows good DM control over the last few months

Lid Lag, Lid retraction, goitre

Graves Disease

Hypothyroidism, no goitre

Amiodarone induced hypothyroidism

Hypertension, striae, moon face

Cushings disease/ syndrome: Remember this fact…

Cushing’s syndrome is the syndrome encompassing all the different clinical entities

Cushing’s disease is the specific release of ACTH from the pituatory itself, thus Cushings disease is one form of the Cushing’s syndrome.

Other causes of Cushings syndrome include: adrenal adenoma

Adrenal carcinoma

Glucocorticoid treatment (e.g. prednisolone in rheumatoid arthritis)

Extreme episodic palpitations with hypertension

Phaeochromocytoma: Tumour that produces catecholamines leading to hypertension, stroke, palpitations and cardiac dysrythmias. Treatment is excision. Diagnosis is based on finding elevated levels of urinary catecholamines followed by imaging of the adrenal glands themselves.

Excessive thirst, normal blood sugar and elecrolytes

Diabetes insipidus: caused by impaired secretion of Anti diuretic hormone (ADH) leading to impaired water retention.

ADH acts exactly as you would expect it to so if you don’t have enough “anti diuretic” the result is diuresis!

Blood tests can be normal. Remember poorly concentrated urine in water depravation can lead to a diagnosis (water depravation test)

Large tongue, prominent supra orbital ridge, changing facial appearance

Acromegaly: Pituatory excessive secretion of growth hormone. Biochemical diagnoses remember IGF1 (insulin like growth factor 1). Gold standard diagnosis is to perform an oral glucose tolerance test and then measure levels of growth hormone.

Paroxsysmal flushing of the face and weight loss

Carcinoid syndrome: excessive release of 5HIAA by this tumour causes paroxsymal flushing and sweats. Diagnosis: 24 hour urine collection for 5HIAA (5 hydroxyindolacetic acid)

Gynecomastia

Marijuana use: other causes include chronic liver disease (alcoholism), testosterone replacement (bodybuilders using drug to combat impotence), other drugs (classically spirinolactone and digoxin)

Pigmented palmar creases, hypothermia

Addison’s disease: Steroid lack secondary to adrenal gland failure of any cause (autoimmune/ cancer infiltration/ TB etc)

Goitre with hypothyroidism

Hashimoto’s thyroiditis: autoimmune infiltration and destruction of the thyroid gland

JVP cannon waves

A 62 year old man has ischaemic heart disease. On inspection of the JVP cannon waves are seen. This is because of

  • Atrial Fibrillation
  • Congestive cardiac failure
  • Ventricular Tachycardia
  • Asystole
  • Complete heart block

Complete heart block

In CHB you get asychronous contraction of the atria and ventricles. Common causes include conduction system disease (IHD) and drugs (e.g. combinations of beta blockers and calcium channel blockers).

Deep breaths

A 22 year old female is sat in an A&E triage cubicle talking to her partner. You observe

She has a respiratory rate of 28
She is taking deep breaths in
She does not appear to be wheezy
She does not look well

The A&E cardex suggests she started a new medicine this morning.

What is being observed?

  • Kussmaul sign
  • Traub’s sign
  • Adverse drug reaction
  • Anaphylaxis
  • Respiratory acidosis
  • Acute asthma

Kussmaul sign

“Kussmaul breathing” – respiratory compensation for a metabolic acidosis.

Traub’s sign is pistol shot femorals on auscultaion of the femorals in sever AR: the nosie as the blood refluxes back through the femoral artery!

Deep breaths 2

Continued from previous question: [A 22 year old female is sat in an A&E triage cubicle talking to her partner. You observe

She has a respiratory rate of 28
She is taking deep breaths in
She does not appear to be wheezy
She does not look well

The A&E cardex suggests she started a new medicine this morning.

What is her most likely underlying diagnosis from the following list

  • Aspirin Overdose
  • Methanol ingestion
  • Alcohol Poisoning
  • Traub’s sign
  • Type I diabetes
  • None of the above

Type I diabetes

Aspirin OD – yes this would do it
Methanol poisoning – yes this would also do it
Lactic acidosis from pancreatitis – yes this would do it

but…. they’re all much rarer than type I diabetes mellitus presenting in this way (diabetic ketoacidosis).

Respiratory matching condition

Match the following features with their underlying condition.

Conditions:

Asthma, Bronchiectasis, COPD, Lobar Collapse, Pneumonia, Pneumothorax, Pleural Effusion, Pulmonary Fibrosis, Pulmonary Embolism, Stridor.

Features:

Expansion

Air entry

Percussion

Auscultation

Vocal resonance

Reduced affected side

Reduced affected side

Hyper-resonant

Reduced air entry

Reduced

Reduced both sides & hyperinflated

Reduced bilaterally

Hyper-resonant

Wheeze+/-\: expiratory polyphonic

Reduced

Reduced both sides

Reduced bilaterally

No change / Reduced

Crackles-fine inspiratory DO NOT clear on coughing

No change / Reduced

Reduced both sides

Reduced bilaterally

No change / Reduced

Crackles-fine inspiratory DO clear on coughing

No change / Reduced

Reduced both sides during active disease

?both sides during active disease

Hyper-resonant

Wheeze\: expiratory polyphonic

No change

Reduced both sides

?both sides

No change

Wheeze\:inspiratory often monophonic

No change

Reduced side of collapse(ipsilaterally), trachea pulled to that side

Reduced ipsilaterally

Reduced ipsilaterally

Reduced air entry

Reduced

Reduced side of collapse

Reduced ipsilaterally

Reduced ipsilaterally

Bronchial breathing, Reduced air entry

Reduced

No change

No change

No change (Reduced if infarcted tissue)

Possible pleural rub (crunching through snow) often heard over area

No change

Reduced side of effusion (ipsilaterally), trachea pushed to other side

Reduced ipsilaterally

Reduced ipsilaterally, stoney dull

Reduced air entry

Reduced

 The findings are as follows for the following conditions…

Condition

Expansion

Air entry

Percussion

Auscultation

Vocal resonance

Pneumothorax

Reduced affected side

Reduced affected side

Hyper-resonant

Reduced air entry

Reduced

COPD

Reduced both sides & hyperinflated

Reduced bilaterally

Hyper-resonant

Wheeze+/-\: expiratory polyphonic

Reduced

Pulmonary fibrosis

Reduced both sides

Reduced bilaterally

No change / Reduced

Crackles-fine inspiratory DO NOT clear on coughing

No change / Reduced

Bronchiectasis

Reduced both sides

Reduced bilaterally

No change / Reduced

Crackles-fine inspiratory DO clear on coughing

No change / Reduced

Asthma

Reduced both sides during active disease

?both sides during active disease

Hyper-resonant

Wheeze\: expiratory polyphonic

No change

Stridor

Reduced both sides

?both sides

No change

Wheeze\:inspiratory often monophonic

No change

Lobar Collapse

Reduced side of collapse(ipsilaterally), trachea pulled to that side

Reduced ipsilaterally

Reduced ipsilaterally

Reduced air entry

Reduced

Pneumonia

Reduced side of collapse

Reduced ipsilaterally

Reduced ipsilaterally

Bronchial breathing, Reduced air entry

Reduced

Pulmonary Embolism

No change

No change

No change (Reduced if infarcted tissue)

Possible pleural rub (crunching through snow) often heard over area

No change

Pleural Effusion

Reduced side of effusion (ipsilaterally), trachea pushed to other side

Reduced ipsilaterally

Reduced ipsilaterally, stoney dull

Reduced air entry

Reduced